Suffering from a chronic disease like psoriasis is often a challenge to stay active and lead a dynamic daily life. Yet, both physical and intellectual activities are one of the main keys to minimize the symptoms.
Suffering from a chronic disease like psoriasis is often a challenge to stay active and lead a dynamic daily life. Yet, both physical and intellectual activities are one of the main keys to minimize the symptoms. Many sources, including the National Psoriasis Foundation, assert that exercise help reduce body inflammation. Also, sport activity and mental focus lower stress level, which is essential to manage any psychosomatic disease.
1 - Find your own pace
It is useless to do too much or put all your efforts in the first days. This would lead to nothing except physical exhaustion. The important thing is to get a steady pace that is suitable for you.
2 - Do not neglect hydratation
It might seem trivial when talking about activities and sport, but a skin affected by psoriasis is constantly in need for water. This is all the more significant as psoriasis causes cutaneous itch, dryness and inflammation.
3 - Try not to stay alone
Exercising with someone sharing your goals is an excellent way to maintain motivation and regularity. This is also a great solution to avoid skipping the sessions or the training you are used to plan.
4 - Wear appropriate clothes
The mix between sweat and materials such as spandex raises the risk of irritation. Consequently, one must always be mindful of wearing risk-free clothes made of, for instance, modal fabric or cotton.
5 - Have a cold shower
Like in many skin chronic diseases, sweat can aggravate the symptoms and using hot water is not an effective way to eliminate perspiration. This is the reason why having a cold shower quickly after a workout is the best option. Not only will you get rid of the sweat, but you will also cool your body, two significant conditions to avoid the symptoms.
6 - If you are afraid of flare-ups
Paradoxically, sport and activities can also generate stress for some people for various reasons (fear of sweating for people affected by a skin disease, fear of injuries, fear of not doing well etc.). These three exercices, known for minimizing the risks generating these anxieties, are consequently considered "safe" for people living with psoriasis :
Sporadic Activities (walking, playing fetch, biking etc.)
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Recent publications on Psoriasis and Atopic Dermatitis
IL-33 contributes to disease severity in Psoriasis-like models of mouse.
Cytokine. 2019 Jul , 119:159-167.
Immune cells infiltrating the psoriatic skin secrete high amounts of pro-inflammatory cytokines IL-17, TNF-α, IL-21 and IL-36 resulting in chronic inflammation. However, the exact cellular and molecular mechanisms have not been fully understood. We report here elevation of IL-33 expression in psoriatic lesions. Studies in imiquimod (IMQ)-induced mice with psoriatic inflammation confirmed a critical role for IL-33 in driving the disease. IL-33 reduces the CD4 and CD8 cells, inhibits autophagy (...)see on pubmed
Association of plasma and urine metals levels with kidney function: A population-based cross-sectional study in China.
Chemosphere. 2019 Jul , 226:321-328.
Although environmental exposure to multiple metals is common, epidemiological studies on the associations of exposure to 23 metals with kidney function have not been analyzed. We aimed to investigate the associations of 23 metals levels with renal function.see on pubmed
Models of human psoriasis: Zebrafish the newly appointed player.
Dev. Comp. Immunol.. 2019 Aug , 97:76-87.
Psoriasis is a human chronic, immune disease with severe cutaneous and systemic manifestations. Its prevalence, among the world population, highly varies with ethnicity and geography, but not sex from remarkable low levels in Asia to 2.3% in Spain, or an impressive 11.5% in Norway. The pathogenesis of psoriasis derives from complex genetic and environmental interactions, which creates aberrant crosstalk between keratinocytes and variated immune cell, resulting in open amplified inflammatory (...)see on pubmed
What's new in atopic eczema? An analysis of systematic reviews published in 2016. Part 3: nomenclature and outcome assessment.
Clin. Exp. Dermatol.. 2019 Jun , 44, (4):376-380.
This review forms part of a series of annual updates that summarize the evidence base for atopic eczema (AE). It presents the key findings from 11 systematic reviews published in 2016 that focus on AE outcome assessment, disease impact and nomenclature. Systematic reviews on the treatment and prevention of AE are summarized in Part 1 of this update, and systematic reviews on the epidemiology of and risk factors for AE are summarized in Part 2. Six reviews summarized what outcome measurement (...)see on pubmed
Discovery of a class of highly potent Janus Kinase 1/2 (JAK1/2) inhibitors demonstrating effective cell-based blockade of IL-13 signaling.
Bioorg. Med. Chem. Lett.. 2019 Jun 15, 29, (12):1522-1531.
Disruption of interleukin-13 (IL-13) signaling with large molecule antibody therapies has shown promise in diseases of allergic inflammation. Given that IL-13 recruits several members of the Janus Kinase family (JAK1, JAK2, and TYK2) to its receptor complex, JAK inhibition may offer an alternate small molecule approach to disrupting IL-13 signaling. Herein we demonstrate that JAK1 is likely the isoform most important to IL-13 signaling. Structure-based design was then used to improve the (...)see on pubmed
Effect of cinnamamides on atopic dermatitis through regulation of IL-4 in CD4 cells.
J Enzyme Inhib Med Chem. 2019 Dec , 34, (1):613-619.
This study aimed to evaluate the effects of cinnamamides on atopic dermatitis (AD) and the mechanisms underlying these effects. To this end, the actions of two cinnamamides, (E)-3-(4-hydroxyphenyl)-N-phenylethyl acrylamide (NCT) and N-trans-coumaroyltyramine (NCPA), were determined on AD by orally administering them to mice. Oral administration of the cinnamamides ameliorated the increase in epidermal and dermal thickness as well as mast cell infiltration. Cinnamamides suppressed serum (...)see on pubmed